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Figure 6: A-Lead poisoning. Case 22: (a and b) axial NCCT showing subcortical calcification at the grey-white junction (arrows-A) and bilateral cerebellar white matter (arrows-B). (c): Hyperglycemia induced hemiballismus and hemichorea. Case 23: (c) axial T1 showing left putaminal hyperintensity (arrow). (d-h): dextropropoxyphene toxicity. Case 24: (d and e) axial FLAIR and T2 showing symmetrical basal ganglia hyperintensity (arrow); (f) axial DWI shows restriction in bilateral caudate head and putamina (arrows); (g) coronal T2 shows symmetrical basal ganglia hyperintensity (arrows); (h) Post-contrast T1 image reveals no enhancement

Figure 6: A-Lead poisoning. Case 22: (a and b) axial NCCT showing subcortical calcification at the grey-white junction (arrows-A) and bilateral cerebellar white matter (arrows-B). (c): Hyperglycemia induced hemiballismus and hemichorea. Case 23: (c) axial T1 showing left putaminal hyperintensity (arrow). (d-h): dextropropoxyphene toxicity. Case 24: (d and e) axial FLAIR and T2 showing symmetrical basal ganglia hyperintensity (arrow); (f) axial DWI shows restriction in bilateral caudate head and putamina (arrows); (g) coronal T2 shows symmetrical basal ganglia hyperintensity (arrows); (h) Post-contrast T1 image reveals no enhancement