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Figure 5: (a-d): CO poisoning. Case 17: (a) axial T2 shows bilateral pallidal hyperintensity (arrows). Case 18: (b) axial T2 shows symmetrical globus pallidus hyperintensity (arrows); (c and d) axial DWI shows restriction in B/L centrum semiovale, subcortical white matter, corpus callosum, and globus pallidus (arrows). (e-h) methyl iodide toxicity. Case 19: (e-g) axial FLAIR showing symmetrical hyperintensity in bilateral dentate nuclei (long arrows-E), central tegmental tracts (short arrows-E), dorsal pons and SCP (arrows-F), posterior lentiform nuclei (arrows-G); (h) sagittal T2 showing long tract hyperintensity in the dorsal brainstem (arrow)

Figure 5: (a-d): CO poisoning. Case 17: (a) axial T2 shows bilateral pallidal hyperintensity (arrows). Case 18: (b) axial T2 shows symmetrical globus pallidus hyperintensity (arrows); (c and d) axial DWI shows restriction in B/L centrum semiovale, subcortical white matter, corpus callosum, and globus pallidus (arrows). (e-h) methyl iodide toxicity. Case 19: (e-g) axial FLAIR showing symmetrical hyperintensity in bilateral dentate nuclei (long arrows-E), central tegmental tracts (short arrows-E), dorsal pons and SCP (arrows-F), posterior lentiform nuclei (arrows-G); (h) sagittal T2 showing long tract hyperintensity in the dorsal brainstem (arrow)