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Figure 1: (a and b) Initial magnetic resonance imaging (MRI) of the brain; fluid-attenuated inversion recovery (FLAIR) and T2-weighted images (day 2 of illness) revealed asymmetrical abnormal signal intensity involving bilateral frontal, precentral, and central subcortical white matter, as well as deep gray and white matter, bilateral ventrolateral thalami, and in left midbrain. (c and d) MRI of the brain; FLAIR and T2 weighted images (day 28 of illness) revealing scarred cortices with large area of gliosis in bilateral frontoparietal perirolandic cortical and subcortical white matter as well as in bilateral thalamocapsular region

Figure 1: (a and b) Initial magnetic resonance imaging (MRI) of the brain; fluid-attenuated inversion recovery (FLAIR) and T2-weighted images (day 2 of illness) revealed asymmetrical abnormal signal intensity involving bilateral frontal, precentral, and central subcortical white matter, as well as deep gray and white matter, bilateral ventrolateral thalami, and in left midbrain. (c and d) MRI of the brain; FLAIR and T2 weighted images (day 28 of illness) revealing scarred cortices with large area of gliosis in bilateral frontoparietal perirolandic cortical and subcortical white matter as well as in bilateral thalamocapsular region